physio biochem doubts

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what sensory unit is responsible for tone of skeletal muscle?
1.muscle spindle
2.golgi tendon
3 sarcomere
4pacinian corpuscles.
this is in 2002unreleased paper..



what best describes hydroxyapatite.
1.it has 12 ions per cell
2solubility decreases as H ion increases
3.it has an amphilic surface
4.it has no ion substitutions in bone and enamel
5.it has higher solubility product constant than fluorapatite



which enzyme does not need a primer..
1.dna ligase.
2 rna polymerase
3.dna polymerases 1 , 2 and 3.

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hi friends,
wat increases the strength of heart muscle contractn-
increase e/cellular calcium or increases i/cellular calcium...
decks say e/cellular increase in calcium....is it rigt...
im confused....wont calcium go i/cellularly to increase the actin myosin cross bridging??????????
sum1 please explain....
thanks...

hi.. its pretty simple... just like chain rxn. 1 unit of e/cellular ca will enter the myocardial cells and cause multiple units of i/cellular ca to be rleased fr SR...

so whn u increase the e/cellular ca, automatically leads to increase in numerous i/cellular ca n M.CONt.however e/cellular ca is NEEDED for the release of intracellular ca.
 
what most immeiately leads to dissociation of actomyosin complex-
1,atp hydrolysis
2.release of atp hydrolytic products
3.atp replacing adp on myosin heads
4.tilting of myosin heads.

not sure...its either 1 or 3

i think its 3.

simply, coz... actinomyosin complex is disloged only when the adp+pi bound to AMcomplex is replaced by ATP molecule...
 
most of the calcuim in human body is excreted through sweat.

coronary artries are dilated maximally (filled with blood) during early diastole. 70% of coronary vessels are filled during diastole and 30% during systole.

most of calcium excretion is through feces ( 1:0.47 ration of feces and rest of the body)
and i read somewhere its late diastole during which there is maximum filling.
 
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hi..wats the difference between oxygen tension and oxygen content of arterial blood?
in anemia, both vl be reduced????????????or one of them????
thats a board question....


I anemia only oxygen content ll be reduced not the tension.

oxygen content of the gas depends soley on the oxygen carrying capcity of rbcs.
 
hey guys..
thanx so much for the answers ....
so, in anemia, o2 tension reduces but o2 content is unchanged..rite????
also,
what is true about g proteins..
1.theyr membrane proteins.
2 they r tightly bound to adenyl cyclase
3 they act on steroid hormones
4.catalyse diacylglycerol production


also, how is arterial compliance reduced by stressful experience????

Gproteins are transmembrane proteins consisting of alpha/beta/gamma subunits. whn an hormone binds to a recptor on cell surface via gprotein mechanism.. it causes confromational change in the gprotein causin the alpha subunit to dissociate frm the remain 2 subunits. the alpha sunbuits binds to gtp.. this causes activation of andynalte cyclase in cell membrane which activates/inhibits camp finally to release a secondary messenger that brings about the desired effect in cell.
 
ph of soln A is 7 and of soln B is 6.
what is true of both solns-
1.soln A HAS 1/10 H IONS THAN B
2.SOLN A HAS 10 TIMES H IONS THAN SOLN B
3 .SOLN A HAS 6/7 H IONS
4 SOLN A HAS 7/6 H IONS




1.soln A HAS 1/10 H IONS THAN B is correct as change in 1 ph unit reuth in 10 fold change in the acid and base ratio.
 
what most immeiately leads to dissociation of dissociation of -
1,atp hydrolysis
2.release of atp hydrolytic products
3.atp replacing adp on myosin heads
4.tilting of myosin heads.

3 is correct .....atp hydrolysis reset the myosin head for next cycle.......atp replacing adp result in immeiate dissociation.
 
most of calcium excretion is through feces ( 1:0.47 ration of feces and rest of the body)
and i read somewhere its late diastole during which there is maximum filling.


the question asks about whn are the CORONARY VESSELS DILATED/FILLED.so its the early diastole where maximum filling takes place i am mentioning this information after having read frm sherwood.. there is a graph plotted in this book to expalin the filling of coronary vessels during the entire diastole.. with a huge bump at early systole. i think i am pretty sure.pls lemme know the source of u r info.
 
oral tuberculosis vl occur by which route-
1.primary inoculation by contaminated instruments
2.sputum entering the mouth
3lymph spread
4 hematogenous spread.


answer 4: hematogenous spread. primary tuberculosis only the LUNG is the site. desimination would result in various sites like bone/breast/ brain/ oral region.

sputum entering the mouth is wrong answer coz of the potent antimicrobial n defense mechanisms of salive which prevent colonization. nasopharnx could be the route only in primary by inspiring infectious droplets.
 
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d amino acids and n acetyl muramic acids are found in which of the following?
1.outer membrane of gram -ve organisms
2.teichoic acid of gram +ve
3.lipopolysaccharide of gram -ve
4.cytoplasmic membrane of gram +ve
5.peptidoglycan of gram -ve and +ve organisms
 
d amino acids and n acetyl muramic acids are found in which of the following?
1.outer membrane of gram -ve organisms
2.teichoic acid of gram +ve
3.lipopolysaccharide of gram -ve
4.cytoplasmic membrane of gram +ve
5.peptidoglycan of gram -ve and +ve organisms

god help me
 
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