physio biochem doubts

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what sensory unit is responsible for tone of skeletal muscle?
1.muscle spindle
2.golgi tendon
3 sarcomere
4pacinian corpuscles.
this is in 2002unreleased paper..



what best describes hydroxyapatite.
1.it has 12 ions per cell
2solubility decreases as H ion increases
3.it has an amphilic surface
4.it has no ion substitutions in bone and enamel
5.it has higher solubility product constant than fluorapatite



which enzyme does not need a primer..
1.dna ligase.
2 rna polymerase
3.dna polymerases 1 , 2 and 3.

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what sensory unit is responsible for tone of skeletal muscle?
1.muscle spindle(ans)
2.golgi tendon
3 sarcomere
4pacinian corpuscles.
this is in 2002unreleased paper..



what best describes hydroxyapatite.
1.it has 12 ions per cell
2solubility decreases as H ion increases
3.it has an amphilic surface
4.it has no ion substitutions in bone and enamel
5.it has higher solubility product constant than fluorapatite(ans)



which enzyme does not need a primer..
1.dna ligase.
2 rna polymerase(ans)
3.dna polymerases 1 , 2 and 3.


i agree with armorshell answers:)
 
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i agree wid mattew ....muscle spindle is responsibl 4 static nd dynamic changes in ms length nd golgi tendon is responsibl 4 muscle tension
 
i agree wid mattew ....muscle spindle is responsibl 4 static nd dynamic changes in ms length nd golgi tendon is responsibl 4 muscle tension

Tension and tonus are two completely different things. Tonus is maintaining a state of constant partial contraction, modified by stretch receptor reflexes, which occur through the muscle spindle. Length of a muscle indirectly determines it's contractile state.

In my understanding, GTOs can only measure changes in force, not constant partial contraction like tone.
 
hi..yes..but the answer key says that its sarcomere...but i feel its muscle spindles..2002 answer key
 
thanks for ur input....i guess the answer key is made by a student...
one more doubt..
predominant route of excretion of calcium...
1.urine
2,feces
3 sweat...
i read that 5 mmols of ca r excreted by kidneys and the same amt by feces....

coronary artery vl undergo maximum dilatation during which phase-
1,early systole
2 late systole
3 early diastole
4 late diastole..
 
ph of soln A is 7 and of soln B is 6.
what is true of both solns-
1.soln A HAS 1/10 H IONS THAN B
2.SOLN A HAS 10 TIMES H IONS THAN SOLN B
3 .SOLN A HAS 6/7 H IONS
4 SOLN A HAS 7/6 H IONS


G PROTEIN ACTIVATION NEEDS-
1 ALPHA SUBUNIT TO BIND GTP
2.ALPHA SUB UNIT TO BIND GTP
3HYDROLYSIS OF BETA -GAMMA SUBUNITS.
4.ALPHA SUB UNITS TO PHOSPHORYLATE DOWNSTREAM TARGETS
 
what most immeiately leads to dissociation of actomyosin complex-
1,atp hydrolysis
2.release of atp hydrolytic products
3.atp replacing adp on myosin heads
4.tilting of myosin heads.
 
hi..wats the difference between oxygen tension and oxygen content of arterial blood?
in anemia, both vl be reduced????????????or one of them????
thats a board question....
 
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ph of soln A is 7 and of soln B is 6.
what is true of both solns-
1.soln A HAS 1/10 H IONS THAN B
2.SOLN A HAS 10 TIMES H IONS THAN SOLN B
3 .SOLN A HAS 6/7 H IONS
4 SOLN A HAS 7/6 H IONS


G PROTEIN ACTIVATION NEEDS-
1 ALPHA SUBUNIT TO BIND GTP
2.ALPHA SUB UNIT TO BIND GDP
3HYDROLYSIS OF BETA -GAMMA SUBUNITS.
4.ALPHA SUB UNITS TO PHOSPHORYLATE DOWNSTREAM TARGETS

correct if m wrong...
 
hi..wats the difference between oxygen tension and oxygen content of arterial blood?
in anemia, both vl be reduced????????????or one of them????
thats a board question....
o2 content is da total o2 present in blood both bound nd dissolved therefore it will nt reduce in anemia......correct if m wrong
 
what most immeiately leads to dissociation of actomyosin complex-
1,atp hydrolysis
2.release of atp hydrolytic products
3.atp replacing adp on myosin heads
4.tilting of myosin heads.

not sure...its either 1 or 3
 
what most immeiately leads to dissociation of actomyosin complex-
1,atp hydrolysis
2.release of atp hydrolytic products
3.atp replacing adp on myosin heads
4.tilting of myosin heads.

not sure...its either 1 or 3

ITS 3
 
hey guys..
thanx so much for the answers ....
so, in anemia, o2 tension reduces but o2 content is unchanged..rite????
also,
what is true about g proteins..
1.theyr membrane proteins.
2 they r tightly bound to adenyl cyclase
3 they act on steroid hormones
4.catalyse diacylglycerol production


also, how is arterial compliance reduced by stressful experience????
 
thanks for ur input....i guess the answer key is made by a student...
one more doubt..
predominant route of excretion of calcium...
1.urine
2,feces
3 sweat...
i read that 5 mmols of ca r excreted by kidneys and the same amt by feces....

coronary artery vl undergo maximum dilatation during which phase-
1,early systole
2 late systole
3 early diastole
4 late diastole..
hii
 
cortisol acts on which enzyme in the liver??
1.phosphofructokinase
2.glucokinase
3pyruvate kinase
4pyruvate carboxylase
 
oral tuberculosis vl occur by which route-
1.primary inoculation by contaminated instruments
2.sputum entering the mouth
3lymph spread
4 hematogenous spread.
 
the most common route of spread of chickenpox is
1 direct skin to skin contact
2 air borne droplets.
i feel it spreads thru both routes..which is the right naswer????
 
Secretory Iga Acts By-
1.lysing Oral Bacteria
2 Phagocytosis Of Bacteria By Oral Leukocytes
3.prevent Adherence Of Bacteria.
 
all the foll cause malabsorption except-
1crohns disease
2 ulcerative colitis
3 giardiasis
4obstructive jaundice
 
best way to sterilize burs-non corrosive-
1 autoclave
2.chemical vapour
3iodop[hors
4ultrasonic cleaning
5 quartenary compounds
 
cortisol acts on which enzyme in the liver??
1.phosphofructokinase
2.glucokinase
3pyruvate kinase
4pyruvate carboxylase
 
hi..
thanx so much for the se answers
but will autoclaving not corrode te burs???
and chickenpox answer is by skin or resp droplets??
pls confirm tese answers and let me know..
thanx n take care...
 
hi friends,
wat increases the strength of heart muscle contractn-
increase e/cellular calcium or increases i/cellular calcium...
decks say e/cellular increase in calcium....is it rigt...
im confused....wont calcium go i/cellularly to increase the actin myosin cross bridging??????????
sum1 please explain....
thanks...
 
hi friends,
wat increases the strength of heart muscle contractn-
increase e/cellular calcium or increases i/cellular calcium...
decks say e/cellular increase in calcium....is it rigt...
im confused....wont calcium go i/cellularly to increase the actin myosin cross bridging??????????
sum1 please explain....
thanks...



that's right, increased e/ cellular Ca wil inc i/cellular & thus inc cross-bridging & so more contraction.
 
hi,
thanx for the answer and ur post on ur nbde experience..
realli helpful..am giving the exam in 2 weeks....
and all confused rite now...
lot of people r saying to read decks and old papers....is it gud enuf????
i dnt hv time for kaplan rebiew book...and r old paper questions frm 1979 onwards repeated or only new papers r important???
 
best way to sterilize burs-non corrosive-
1 autoclave
2.chemical vapour
3iodop[hors
4ultrasonic cleaning
5 quartenary compounds

the answer definitely is 2. chemical vapor (ethylene oxide). autoclave will dull/corrode. got this from Kaplan review and First Aid books.
 
hi friends,
wat increases the strength of heart muscle contractn-
increase e/cellular calcium or increases i/cellular calcium...
decks say e/cellular increase in calcium....is it rigt...
im confused....wont calcium go i/cellularly to increase the actin myosin cross bridging??????????
sum1 please explain....
thanks...

calcium induced calcium release seen in heart.same voltage that open fast gated sodium gates open also opens slow calcium gated,this calcium goes in and release calcium from SR.???
 
calcium induced calcium release seen in heart.same voltage that open fast gated sodium gates open also opens slow calcium gated,this calcium goes in and release calcium from SR.???

first of all g protein is transmembrane protein what span around the membrane 7 times. it is activates when alfa subunit is bounded to gtp and deactivated with gdp.

once ap arrives the heart it depolerize the membrane..
at upstroke phase fast na channel are open and also ca2+..
the thing is that na are fast open and fast close..this is why the upstoke effect is short and powerful.ca2+ channels are open at the same time but because they are slow acting the effect is seen only at the plateu phase then na are in inactvated phase and when k+ are open as well (balancing effect)

btw there r 2 types of k+ channels. 1 is independent of ligand and its open all the time (this why membrane potential is more negative -90,if u compare it skeletal or neuro membrane potential ) the 2nd type is slow k+ that are open together with na and ca2+ abd the effect can be seen at the plateu phase where only slow ca+ and K are open,

correct me if im wrong
 
in the ph question shouldnt it be that the solution A will have 10 times the H ions as soln B. pH 7 means 1 X 10 raise to power 7 ions
 
first of all g protein is transmembrane protein what span around the membrane 7 times. it is activates when alfa subunit is bounded to gtp and deactivated with gdp.

once ap arrives the heart it depolerize the membrane..
at upstroke phase fast na channel are open and also ca2+..
the thing is that na are fast open and fast close..this is why the upstoke effect is short and powerful.ca2+ channels are open at the same time but because they are slow acting the effect is seen only at the plateu phase then na are in inactvated phase and when k+ are open as well (balancing effect)

btw there r 2 types of k+ channels. 1 is independent of ligand and its open all the time (this why membrane potential is more negative -90,if u compare it skeletal or neuro membrane potential ) the 2nd type is slow k+ that are open together with na and ca2+ abd the effect can be seen at the plateu phase where only slow ca+ and K are open,

correct me if im wrong
do you think i am wrong?
 
no no u were right i was just supporting ur answer.. at the bottom line ca2+ enter from extracellular and cause a release from sr to adittional ca2
sorry if i made u doubt ur answer :)
 
i dont have any doubt in am 100% sure that i am correct:D
sorry if i made u doubt ur answer :)[/quote]
 
thanks for ur input....i guess the answer key is made by a student...
one more doubt..
predominant route of excretion of calcium...
1.urine
2,feces
3 sweat...
i read that 5 mmols of ca r excreted by kidneys and the same amt by feces....

coronary artery vl undergo maximum dilatation during which phase-
1,early systole
2 late systole
3 early diastole
4 late diastole..

most of the calcuim in human body is excreted through sweat.

coronary artries are dilated maximally (filled with blood) during early diastole. 70% of coronary vessels are filled during diastole and 30% during systole.
 
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