Let's try again, join me for study group NBDE I may/June 2010

[blissonearth]

Hi friends, let's continue the closed thread here. No promoting any materials here, just prop some in doubt questions as our exam is approaching fast.

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[georgia doc]

differnce betn hemangioma and lymphangioma ,clinically..........how will u differentiate these 2

answer...............


http://www.lpch.org/DiseaseHealthInfo/HealthLibrary/craniofacial/vascmalf.html

 

[georgia doc]

and white spongy nevus and mumucous membrane pemphigoid......

white sponge nevus:
cause: mutation of keratin and keratin genes
autosomal dominant
benign but often mistaken for leukoplakia
presentation: thick, bilateral white plaque with spongy texture
site: buccal mucosa, sometimes labial mucosa ,alveolar ridge or floor of mouth
but never on gingival margin and dorsum of tongue.........


mucous membrane pemphigoid:
also called cicatrial pemphigoid or benign mucous membrane pemphigoid
incidence: rare ,chronic autoimmune sub epithelial blistering ( at basement membrane level)
presentation: erosive, scarring of some involved parts of skin and mucous membrane
site: in mouth,lesions on gingiva................elsewhere in body,such as sinuses,genital.

 

[wdent]

Hey pb2007 and asheer ...
For curiosity i looked at ur question about bennett mvt in nbde ist aid it says that : occur during LATERAL excursive movements in which WORKING side condyle bodily shifts LATERAL(toward working side )


Asheer why it is downward forward and MEDIAL?
Havnt started with D.A yet !just interested to know it ?

 

[pb2007]

Hey pb2007 and asheer ...
For curiosity i looked at ur question about bennett mvt in nbde ist aid it says that : occur during LATERAL excursive movements in which WORKING side condyle bodily shifts LATERAL(toward working side )


Asheer why it is downward forward and MEDIAL?
Havnt started with D.A yet !just interested to know it ?

hi wadent ,
bennett movement can be measured both in working and non workin condyles but it is usually done in relation to non working condyl.that's what my textbuk mentions .even bennett angle isformed between the path of non workin condyle and the sagittal plane .
so in non workin pathways the condyle moves forward downward n medially .
for more info read page no 804 of kapaln notes in DA under lateral contacting movement.
plz correct if i'm wrong .

 

[teethie]

hi guys,
lots of discussiongoing on, here is my explanation:

wadent: u r right that bennet movement occur during LATERAL excursive movements in which WORKING side condyle bodily shifts LATERAL(toward working side )

pb2007 : u r right too that bennett angle isformed between the path of non workin condyle and the sagittal plane .

the key lies n two words: movement and angle
so to make it more clear, when we say movement it is on working side
and when we say angle it is formed between nonworking condye and sagittal plane.
hope it is clear now.

 

[aathmashree]

white sponge nevus:
cause: mutation of keratin and keratin genes
autosomal dominant
benign but often mistaken for leukoplakia
presentation: thick, bilateral white plaque with spongy texture
site: buccal mucosa, sometimes labial mucosa ,alveolar ridge or floor of mouth
but never on gingival margin and dorsum of tongue.........


mucous membrane pemphigoid:
also called cicatrial pemphigoid or benign mucous membrane pemphigoid
incidence: rare ,chronic autoimmune sub epithelial blistering ( at basement membrane level)
presentation: erosive, scarring of some involved parts of skin and mucous membrane
site: in mouth,lesions on gingiva................elsewhere in body,such as sinuses,genital.

good expn tnx man,...............actually q it was asked as greyish white pearls on buccal mucosa .so i got confused with both.
tnx

 

[elmos]

does cortisol increased protein anabolism and how?

a dental patient presents with bluish discoloration of the mucous membrane indicative of cyanosis the condition is most likely result?
a decreased hemoglobin concentration
a decreased red blood cell concentration
increased level of reduced hemoglobin
decreased blood flow to the affected area
pls what is the answer and explanation

 

[pb2007]

does cortisol increased protein anabolism and how?

a dental patient presents with bluish discoloration of the mucous membrane indicative of cyanosis the condition is most likely result?
a decreased hemoglobin concentration
a decreased red blood cell concentration
increased level of reduced hemoglobin.................answer
decreased blood flow to the affected area
pls what is the answer and explanation

i think 3rd choice makes sence coz Cyanosis is a blue coloration of the skin and mucous membranes due to the presence of greater than, or equal to, 2.5 g/dL of deoxygenated hemoglobin in blood vessels.
reduced hemoglobin is the one that is not combined with oxygen ,so i think the hemoglobin or rbc concentration is not affected .
plz correct if wrong.
cortisol's primary functions are to increase blood sugar and stores of sugar in the liver as glycogen, aid in fat, protein and carbohydrate metabolism.

 

[georgia doc]

hi guys,
lots of discussiongoing on, here is my explanation:

wadent: u r right that bennet movement occur during LATERAL excursive movements in which WORKING side condyle bodily shifts LATERAL(toward working side )

pb2007 : u r right too that bennett angle isformed between the path of non workin condyle and the sagittal plane .

the key lies n two words: movement and angle
so to make it more clear, when we say movement it is on working side
and when we say angle it is formed between nonworking condye and sagittal plane.
hope it is clear now.

agreed

 

[teethie]

agree with pb2007

cortisol does not cause protein anabolism but it causes protein breakdown and lipolysis.

i think 3rd choice makes sence coz Cyanosis is a blue coloration of the skin and mucous membranes due to the presence of greater than, or equal to, 2.5 g/dL of deoxygenated hemoglobin in blood vessels.
reduced hemoglobin is the one that is not combined with oxygen ,so i think the hemoglobin or rbc concentration is not affected .
plz correct if wrong.
cortisol's primary functions are to increase blood sugar and stores of sugar in the liver as glycogen, aid in fat, protein and carbohydrate metabolism.

 

[elmos]

in unrealeased 2000
cushing's syndrome is characterized by each of the following except:
increased protein anabolism
hyperkalemia
which one is it?
and why? pls

 

[teethie]

hi elmos, this is what i found, though it is not the answer but it is the concept given.

Manifestations of Steroid Excess:
Altered electrolyte metabolism (mineralocorticoid excess)
Excess cortisol interacts with mineralocorticoid receptors leading to:

• Sodium retention causing hypertension (in 70-80%).
• Potassium loss: hypokalemic alkalosis in 20% of cases

so neither there is hyperkalemia nor protein anabolism.
pl correct anyone if i am wrong.

in unrealeased 2000
cushing's syndrome is characterized by each of the following except:
increased protein anabolism
hyperkalemia
which one is it?
and why? pls

 

[elmos]

so the answer is both?

 

[asheer]

hi wadent ,
bennett movement can be measured both in working and non workin condyles but it is usually done in relation to non working condyl.that's what my textbuk mentions .even bennett angle isformed between the path of non workin condyle and the sagittal plane .
so in non workin pathways the condyle moves forward downward n medially .
for more info read page no 804 of kapaln notes in DA under lateral contacting movement.
plz correct if i'm wrong .

hey pb2007 just a lil curiosity i jus wanted to knw which editin is ur kaplan notes cos i don hav it in 804 page abt bennets mov mine is a 2009 edition was feelin if am follwin a wrong book!

 

[pb2007]

hey pb2007 just a lil curiosity i jus wanted to knw which editin is ur kaplan notes cos i don hav it in 804 page abt bennets mov mine is a 2009 edition was feelin if am follwin a wrong book!

what i have is kaplan review book ,it doesnt mention year but i got it around 2007 .i myself actuially dont know if there is any difference between kaplan dent essential and review book.but i was familiar with this edition so never bothered gettin new one .
that page no 804 does not give bennentt movement but it talks in terms of workin n non workin but according to teethie's correction i'm myself confused about this movement n angle thing now .

 

[teethie]

hey guys, i am pasting a link which you can read abt bennet angle and movement.

http://books.google.ca/books?id=-wF...&resnum=4&ved=0CCkQ6AEwAw#v=onepage&q&f=false

 

[elmos]

did anyone here about the usc dental anatomy question of 2009 online
is it useful?

 

[pb2007]

hey guys, i am pasting a link which you can read abt bennet angle and movement.

http://books.google.ca/books?id=-wF...&resnum=4&ved=0CCkQ6AEwAw#v=onepage&q&f=false

thanks teethie ,my doubt is cleared now but hav another ques ,in workin side movement its the condyle that moves in pure lateral movement and non workin side the condyle moves forward downward n medially but as far as teeth are concerned irespective of working or non workin side they show mesio distal movement .so is it because of this reason that we picked 1st choice as answer.???plz confirm if this is the concept behind this ques .
i hope i didnt confuse u .
another ques ..is there any effect of position of central fossa n depth of distal fossa on bennett movement as last two choices suggest .

 

[asheer]

hey thanks pb2007 i uderstand nw mine is the lecture notes so it might be the diffrnce but seriousely thanks fr patntly ans my silly doubt!

 

[teethie]

yes pb2007, this is why answer choice is mesio distal positioning of cusps. i did nott read any relationship between position and depth of fossa n bennett movement.

thanks teethie ,my doubt is cleared now but hav another ques ,in workin side movement its the condyle that moves in pure lateral movement and non workin side the condyle moves forward downward n medially but as far as teeth are concerned irespective of working or non workin side they show mesio distal movement .so is it because of this reason that we picked 1st choice as answer.???plz confirm if this is the concept behind this ques .
i hope i didnt confuse u .
another ques ..is there any effect of position of central fossa n depth of distal fossa on bennett movement as last two choices suggest .

 

[wdent]

1)A dentist was performing treatment on patient,But unexpectly the patient lost his conciousness and his head drop ,which of the following is the Best answer?
a.Asthma
b.Allergic to latex gloves
c.Anapylaxis..............answer ?????!!! is it true
d.Scared of dentist

 

[asheer]

hey ans fr ur qstn-( a) - ans is local lymph nodes!


(b) -ya the most probable ans is anaphylaxis .

 

[wdent]

thanks 4 the confirmation asheer !

 

[aathmashree]

a bacterial mutation leading to requirement for single aa is due to
1.lack of mrna
2.loss of ability to utilise glucose
3.absence of single enzyme activity


can any1 plz explain the ans 222........................

 

[aathmashree]

microbial population of perio pockets is.......
1.capable of producing high neutralising ab titre
2.of low order of intrinsic pathogenicity............ans
are the perio bacteria low pathogenic....is that rt ans

 

[ddsaspi]

Each of the following is commonly a part of CHF except
1 Dyspnea
2 cynaosis
3 anasarca
4 ankle edema
5 passive congestion of the liver,

Which should be the best option

 

[teethie]

Cyanosis which suggests severe hypoxemia, is a late sign of extremely severe pulmonary edema.
http://en.wikipedia.org/wiki/Heart_failure

Each of the following is commonly a part of CHF except
1 Dyspnea
2 cyanosis--------ans
3 anasarca
4 ankle edema
5 passive congestion of the liver,

Which should be the best option

 

[wdent]

Agree with u teethie !

 

[elmos]

Teethie you have to remember that in CHF you have pulmonary edema specially from left heart failure which can cause cyanosis, but anarasca is rarely seen in CHF is seen in kidney failure.

 

[elmos]

the host response to malignancy is best reflected by:
marked cellularity of tumor
many mitotic figures in tumor
lymphocytic infiltration at the edge of the neoplasm
a large number of blood vessels
large areas of necrosis in the center
anyone knows the answer and explanation please.

 

[teethie]

elmos,
can you please post a reference in support of your answer.

Teethie you have to remember that in CHF you have pulmonary edema specially from left heart failure which can cause cyanosis, but anarasca is rarely seen in CHF is seen in kidney failure.

 

[wdent]

Hey elmos ... just wana say somthin anasarca 100% can be caused by CHF....
thanks !

 

[wdent]

the host response to malignancy is best reflected by:
marked cellularity of tumor
many mitotic figures in tumor
lymphocytic infiltration at the edge of the neoplasm..........answer (i think so) ill go with this choice..
a large number of blood vessels
large areas of necrosis in the center
anyone knows the answer and explanation please.[/QUOTE]

Pls correct me if iam wrong !!

 

[aathmashree]

Cyanosis which suggests severe hypoxemia, is a late sign of extremely severe pulmonary edema.
http://en.wikipedia.org/wiki/Heart_failure

even i hav the same doubt regrding this q,
this q has been repeated twice in previous q paper n in both the ans r anasarca.......
actally in chf,there is ankle and sacral oedema ....and anasarca r not always process as a clinical manifestation.

 

[aathmashree]

the host response to malignancy is best reflected by:
marked cellularity of tumor
many mitotic figures in tumor
lymphocytic infiltration at the edge of the neoplasm..........answer (i think so) ill go with this choice..
a large number of blood vessels
large areas of necrosis in the center
anyone knows the answer and explanation please.

Pls correct me if iam wrong !! [/QUOTE]


s the choice is correct

 

[teethie]

thank u aathmashree for the clarification. well, now no debate left, just agreeing with asda key.

even i hav the same doubt regrding this q,
this q has been repeated twice in previous q paper n in both the ans r anasarca.......
actally in chf,there is ankle and sacral oedema ....and anasarca r not always process as a clinical manifestation.

 

[wdent]

Hey teethie/aathmashree..

Ive been doin some research on that quetion,,, well in wikki it says the chf--------->anasrca...
But ill say lets stick with ASDA paper !! ......

 

[aathmashree]

change of more specialised cell type to less specialised cell type is
1.dysplasia
.2.anaplsia
3.metaplasia
i know metaplsia is from 1 cell type to another,but if it means towards less speciatised ,is it towards dedifferentiation n is it anaplasia..correct me

 

[rose82]

change of more specialised cell type to less specialised cell type is
1.dysplasia
.2.anaplsia
3.metaplasia
i know metaplsia is from 1 cell type to another,but if it means towards less speciatised ,is it towards dedifferentiation n is it anaplasia..correct me

Im just inferring from wiki.
Metaplasia is conversion of a specialised pseudostr ciliated to stratified sq.(one cell to another)Here there is loss of epithelial function,but it is adapting to a change.

Anaplasia...same cell is undergoing change to less differentiated form.no conversion from one to another.

Its not clear if the question is asking from one cell to another..or same.I think it is Metaplasia considering a change in q like this
change of a more specialised cell type to a less specialised cell type is

Please correct me as im not sure..i just thought this way.Good q aathmashree

 

[teethie]

it is metaplasia.
anaplasia is total loss of differentitation, there is no conversion.

change of more specialised cell type to less specialised cell type is
1.dysplasia
.2.anaplsia
3.metaplasia
i know metaplsia is from 1 cell type to another,but if it means towards less speciatised ,is it towards dedifferentiation n is it anaplasia..correct me

 

[aiming 4 dds]

can anybody suggest me from where shud i do general histology n oral histology as when i do questions my most of d part goes wrong

 

[georgia doc]

plz answer these questions today!


1.endogenous infection?
.syphilis
.dysentery
.gas gangrene
.scarlet fever
.subacute infective endocarditis
2.striated duct of parotid gland lined with?

. simple cuboidal
.stratified cuboidal
.simple columnar
3.adipose tissue is found in submucosa of palate in
.rugae.......................answer? remember reading it in an old thread,wadent confirmed , i guess? plz wadent, reconfirm
.incisive papilla
.midline at the level of molar
.lateral area at the level of premolar
.lateral area at the level of molars
4.after the hand is immersed in warm water for several minutes, the sensation of heat decreases because
.adaptation of thermal receptor
.two point discrimination
.central inhibition ..................ans??
.kinesthetic sense
5.within thoracic cavity,rami communicantes connect the sympathetic trunk with.
.thoracic spinal nerves.................ans??
.splanchnic nerves
.thoracic viscera
.cardiac nerves

 

[aathmashree]

4.ans. i thnk shud b adaptation for thermal receptors,not the other

 

[asheer]

a- is it 1.endogenous infection?
.syphilis
.dysentery-ans
.gas gangrene
.scarlet fever
.subacute infective endocarditis
2.striated duct of parotid gland lined with?

. simple cuboidal
.stratified cuboidal
.simple columnar-ans

within thoracic cavity,rami communicantes connect the sympathetic trunk with.
.thoracic spinal nerves.................ans??
.splanchnic nerves
.thoracic viscera
.cardiac nerves correct me if am wrong!

 

[aathmashree]

1.endogenous infectn r those from normal floura of body,
so cud b dysentry..correct me
2. parotid duct ans r always different in diff q paper,even i want 2 know the correct ans.....

 

[aathmashree]

which of follong r usual constituents of inflammatory infilterate
1.fluids
2.neutrophils
3.macrophages
4.multinucleated giant cells
ans .as...1.2.and 4 not macrophages

but in chronic inflammation macrophages n giant cell has 2 b there,then y not 3. also



microorganism of which of foll infection require a specifc receptor site
..1.anthrx
2.syphylis
3.influenza.ans
4.dysentry

can any 1 explain y

 

[aathmashree]

hey geiorge,if u hav exams tomarrow
gudluk

 

[rose82]

.endogenous infection?
.syphilis
.dysentery
.gas gangrene
.scarlet fever
.subacute infective endocarditis

I think so because Strept is found in normal oral cavity.Please correct me.

 

[teethie]

aathmashree,i agree with u.

4.ans. i thnk shud b adaptation for thermal receptors,not the other

 

[teethie]

plz answer these questions today!


1.endogenous infection?
.syphilis
.dysentery-can not be the answer it is caused by shigella
.gas gangrene
.scarlet fever
.subacute infective endocarditis--answer