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Long time lurker, first time poster. Been really enjoying all the excellent case discussions on here, so thought I'd jump in with my own question:
Thoracic surgeon at my shop insists he can tell when a patient is "inadequately relaxed" b/c the muscle fibers jump more when he bovies, even though the patient has 0 twitches (checked at facial nerve, which should be even more sensitive for 'inadequate' blockade). I tried explaining to him that no amount of NMB can abolish direct muscle stimulation from a supra-maximal stimulus (monopolar cautery), but he doesn't buy it. Am I missing something here? Obviously the bovie might be stimulating small nerve fibers in the muscle as well, and so in theory the radius of the area that's affected might be larger when neuromuscular transmission is intact, but even with 100% receptors blocked, the muscles still seem to jump when buzzed. He insists otherwise. Any thoughts on this topic? (Whether a surgeon should really need 1000% muscle relaxation for a straightforward VATS case is another topic, but I digress...)
Thoracic surgeon at my shop insists he can tell when a patient is "inadequately relaxed" b/c the muscle fibers jump more when he bovies, even though the patient has 0 twitches (checked at facial nerve, which should be even more sensitive for 'inadequate' blockade). I tried explaining to him that no amount of NMB can abolish direct muscle stimulation from a supra-maximal stimulus (monopolar cautery), but he doesn't buy it. Am I missing something here? Obviously the bovie might be stimulating small nerve fibers in the muscle as well, and so in theory the radius of the area that's affected might be larger when neuromuscular transmission is intact, but even with 100% receptors blocked, the muscles still seem to jump when buzzed. He insists otherwise. Any thoughts on this topic? (Whether a surgeon should really need 1000% muscle relaxation for a straightforward VATS case is another topic, but I digress...)